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dc.creatorBrañes, Maria C
dc.creatorCorvalan, L A
dc.creatorEugenin Arce, Eliseo Alberto
dc.creatorGonzalez, Hernan
dc.creatorMartinez Carrasco, Agustin Demetrio
dc.creatorPalisson Etcharren, Francis
dc.creatorSaez Carreño, Juan Carlos
dc.date.accessioned2016-12-27T21:49:29Z
dc.date.available2016-12-27T21:49:29Z
dc.date.issued2000
dc.identifier.isbn978-012-550-645-8
dc.identifier.urihttp://hdl.handle.net/10533/165245
dc.description.abstractThe properties of the inflammatory response depend on the quality, intensity, and duration of the insult (e.g., microorganism, foreign molecule, trauma, burn, and infarct), as well as on the individual and the affected tissue. Moreover, an inflammatory process could be acute or chronic and it is mediated chiefly by the innate or the specific immune system. The main components of innate immunity are physical and chemical barriers (e.g., epithelia and antimicrobial substances), blood proteins (e.g., complement factors), phagocytic cells (e.g., neutrophils and macrophages), and other leukocytes (e.g., natural killer cells). On the other hand, the principal components of the specific immunity are humoral (antibodies) and cellular (CD4+T cells). The specific immune response amplifies the mechanism of innate immunity and enhances their function, particularly upon repeated exposures to the same foreign antigen. Inflammation is a progressive process that shows overlapping phases. Whereas the acute unspecific response is characterized by hernodynamic, metabolic, and cellular changes, the specific response begins with the recognition of the antigen by specific lymphocytes, followed by their proliferation and differentiation into effector cells.
dc.language.isoeng
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI2.0
dc.relationinstname: Conicyt
dc.relationreponame: Repositorio Digital RI 2.0
dc.relation.ispartofseriesCURRENT TOPICS IN MEMBRANES
dc.titleGAP JUNCTIONS IN INFLAMMATORY RESPONSES: CONNEXINS, REGULATION AND POSSIBLE FUNCTIONAL ROLES
dc.typeCapitulo de libro
dc.countryESTADOS UNIDOS DE AMERICA
dc.bibliographicCitation.stpage555
dc.bibliographicCitation.endpage579
dc.identifier.folio8990008
dc.description.conicytprogramFONDECYT
dc.relation.projectidinfo:eu-repo/grantAgreement/Fondecyt/8990008
dc.relation.setinfo:eu-repo/semantics/dataset/hdl.handle.net/10533/93479
dc.rights.driverinfo:eu-repo/semantics/openAccess
dc.type.driverinfo:eu-repo/semantics/bookPart
dc.description.shortconicytprogramFONDECYT
dc.title.libroGAP JUNCTIONS. MOLECULAR BASIS OF CELL COMMUNICATION IN HEALTH AND DISEASE
dc.creator.libroBenos, Dale
dc.creator.libroPeracchia, Camilo
dc.description.libropages648
dc.publisher.editorialACADEMIC PRESS


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