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dc.coverage.spatialJerusalén
dc.creatorBarahona, María José
dc.creatorRecabal, Antonia
dc.creatorSalgado, Magdiel
dc.creatorOrdenes, Patricio
dc.creatorPalma, A
dc.creatorSepúlveda, Fernando J
dc.creatorGarcía-Robles, María De Los Ángeles
dc.date.accessioned2019-03-27T15:55:31Z
dc.date.available2019-03-27T15:55:31Z
dc.date.issued2017
dc.identifier.urihttp://hdl.handle.net/10533/233040
dc.description.abstractGlucose is a key modulator of feeding behavior both at peripheral tissues and the central nervous system, where it is known to modify neuropeptide expression involved in hunger/satiety signals. In the brain, glucose sensing occurs in the hypothalamus and
dc.language.isoeng
dc.titleHypothalamic GLUT2 inhibition impacts the feeding behavior
dc.typePonencia
dc.identifier.folio1140677
dc.country.isoIsrael
dc.description.conicytprogramFONDECYT
dc.relation.ncongress42°
dc.relation.projectidinfo:eu-repo/grantAgreement/Fondecyt/1140677
dc.rights.driverinfo:eu-repo/semantics/openAccess
dc.date.start2017
dc.date.end2017
dc.description.shortconicytprogramFONDECYT
dc.relation.namecongFederation of European Biochemical Societies Congress - FEBS


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