Browsing Ponencias by Project Id "1100926"

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A Study of CXCR3 and CXCL10 in Papillary Thyroid Cancer

Autor: Bohmwald-Prieto, Karen; Leiva, Andrea; Riquelme, Erick; Véliz, Loreto; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea; González, Hernán Año: 2010N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Altered chemokine receptor expression in papillary thyroid cancer

Autor: Riedel-Soria, Claudia Andrea Año: 2011N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Assesstement of the viability of oligodendrocytes from the progeny gestated in mothers with thyroid hormones deficiency in response to inflammatory molecules

Autor: Albornoz, Eduardo A; Cortes-Pérez, Claudia Mariana; González, Pablo A; Cisternas, Pablo A; Cautivo-Reyes, Kelly Margarita; Carreño-M, Leandro J; Bueno-Ramirez, Susan Marcela; Berman, Joan W; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2011N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Cloning and Characterization of Auliscomys boliviensis Sodium Iodide Symporter

Autor: Arriagada-Serrano, Alejandro Andrés; Miranda, C; Navarro, C; Bueno-Ramirez, Susan Marcela; Molina, A; Gompert, B; Valenzuela, M; Cabello-Guzmán, Gerardo Andrés; Carrasco, Nancy; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2010N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Differential gene expression and altered cell viability induced by inflammatory damage in astrocytes from offspring gestated in hypothyroxinemic mothers

Autor: González, Pablo A; Opazo-Delgado, María Cecilia; Albornoz, Eduardo A; Carreño-M, Leandro J; Bueno-Ramirez, Susan Marcela; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2012N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Gestational thyroid hormone deficiency affects the role of astrocytes on neuronal development, synapsis formation and immunogenicity in the offspring

Autor: Acevedo, K; Zúñiga, G; Cisternas, Pablo A; Flores, B; Opazo-Delgado, María Cecilia; Carreño-M, Leandro J; Boudin, Helene; Bueno-Ramirez, Susan Marcela; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2012N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Hypothyroidism during pregnancy increases the severity of experimental autoimmune encephalomyelitis in the adult offspring

Autor: Albornoz, Eduardo A; Rivero, Tomás; Espinoza, Janyra A; Carreño-M, Leandro J; Cortes-Pérez, Claudia Mariana; González, Pablo A; Cisternas, Pablo A; Cautivo-Reyes, Kelly Margarita; Catalán, Tamara; Eugenin-Arce, Eliseo Alberto; Berman, Joan W; Bueno-Ramire Año: 2012N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Hypothyroidism during Pregnancy Increases the Severity of Experimental Autoimmune Encephalomyelitis in their Adult Offspring

Autor: Albornoz, Eduardo A; Cortes-Pérez, Claudia Mariana; González, Pablo A; Cisternas, Pablo A; Opazo-Delgado, María Cecilia; Eugenin-Arce, Eliseo Alberto; Berman, Joan W; Bueno-Ramirez, Susan Marcela; Carreño-M, Leandro J; Kalergis-Parra, Alexis Mikes; Riedel Año: 2012N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Iodide (I-) Induces a Rapid Inhibition of Na+/I- Symporter (NIS) Through Reactive Oxygen Species

Autor: Arriagada-Serrano, Alejandro Andrés; Becerra, Álvaro; Miranda, C; Plaza, A; Simon, F; Bueno-Ramirez, Susan Marcela; Kalergis-Parra, Alexis Mikes; Carrasco, Nancy; Riedel-Soria, Claudia Andrea Año: 2011N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Maternal thyorid hormone deficiency causes alteration in neurons and astrocytes in their offspring

Autor: Cisternas, Pablo A; González, Pablo A; Zúñiga, G; Cortes-Pérez, Claudia Mariana; Carreño-M, Leandro J; Bueno-Ramirez, Susan Marcela; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2011N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Na+/ I- symporter (NIS) expression and localization during rapid transport inhibition by iodide excess.

Autor: Arriagada-Serrano, Alejandro Andrés; Albornoz, Eduardo A; Bueno-Ramirez, Susan Marcela; Carrasco, Nancy; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2010N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT
Thyroid hormones deficiencies during gestation increase the susceptibility to develop EAE in the offspring

Autor: Cautivo-Reyes, Kelly Margarita; Cortes-Pérez, Claudia Mariana; Albornoz, Eduardo A; González, Pablo A; López, Camila; Carreño-M, Leandro J; Kalergis-Parra, Alexis Mikes; Riedel-Soria, Claudia Andrea Año: 2010N° de proyecto: 1100926Tipo: PonenciaPrograma: FONDECYT

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Browsing Ponencias by Project Id "1100926"

A Study of CXCR3 and CXCL10 in Papillary Thyroid Cancer

Altered chemokine receptor expression in papillary thyroid cancer

Assesstement of the viability of oligodendrocytes from the progeny gestated in mothers with thyroid hormones deficiency in response to inflammatory molecules

Cloning and Characterization of Auliscomys boliviensis Sodium Iodide Symporter

Differential gene expression and altered cell viability induced by inflammatory damage in astrocytes from offspring gestated in hypothyroxinemic mothers

Gestational thyroid hormone deficiency affects the role of astrocytes on neuronal development, synapsis formation and immunogenicity in the offspring

Hypothyroidism during pregnancy increases the severity of experimental autoimmune encephalomyelitis in the adult offspring

Hypothyroidism during Pregnancy Increases the Severity of Experimental Autoimmune Encephalomyelitis in their Adult Offspring

Iodide (I-) Induces a Rapid Inhibition of Na+/I- Symporter (NIS) Through Reactive Oxygen Species

Maternal thyorid hormone deficiency causes alteration in neurons and astrocytes in their offspring

Na+/ I- symporter (NIS) expression and localization during rapid transport inhibition by iodide excess.

Thyroid hormones deficiencies during gestation increase the susceptibility to develop EAE in the offspring

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Browsing Ponencias by Project Id "1100926"

A Study of CXCR3 and CXCL10 in Papillary Thyroid Cancer ﻿

Altered chemokine receptor expression in papillary thyroid cancer ﻿

Assesstement of the viability of oligodendrocytes from the progeny gestated in mothers with thyroid hormones deficiency in response to inflammatory molecules ﻿

Cloning and Characterization of Auliscomys boliviensis Sodium Iodide Symporter ﻿

Differential gene expression and altered cell viability induced by inflammatory damage in astrocytes from offspring gestated in hypothyroxinemic mothers ﻿

Gestational thyroid hormone deficiency affects the role of astrocytes on neuronal development, synapsis formation and immunogenicity in the offspring ﻿

Hypothyroidism during pregnancy increases the severity of experimental autoimmune encephalomyelitis in the adult offspring ﻿

Hypothyroidism during Pregnancy Increases the Severity of Experimental Autoimmune Encephalomyelitis in their Adult Offspring ﻿

Iodide (I-) Induces a Rapid Inhibition of Na+/I- Symporter (NIS) Through Reactive Oxygen Species ﻿

Maternal thyorid hormone deficiency causes alteration in neurons and astrocytes in their offspring ﻿

Na+/ I- symporter (NIS) expression and localization during rapid transport inhibition by iodide excess. ﻿

Thyroid hormones deficiencies during gestation increase the susceptibility to develop EAE in the offspring ﻿

A Study of CXCR3 and CXCL10 in Papillary Thyroid Cancer

Altered chemokine receptor expression in papillary thyroid cancer

Assesstement of the viability of oligodendrocytes from the progeny gestated in mothers with thyroid hormones deficiency in response to inflammatory molecules

Cloning and Characterization of Auliscomys boliviensis Sodium Iodide Symporter

Differential gene expression and altered cell viability induced by inflammatory damage in astrocytes from offspring gestated in hypothyroxinemic mothers

Gestational thyroid hormone deficiency affects the role of astrocytes on neuronal development, synapsis formation and immunogenicity in the offspring

Hypothyroidism during pregnancy increases the severity of experimental autoimmune encephalomyelitis in the adult offspring

Hypothyroidism during Pregnancy Increases the Severity of Experimental Autoimmune Encephalomyelitis in their Adult Offspring

Iodide (I-) Induces a Rapid Inhibition of Na+/I- Symporter (NIS) Through Reactive Oxygen Species

Maternal thyorid hormone deficiency causes alteration in neurons and astrocytes in their offspring

Na+/ I- symporter (NIS) expression and localization during rapid transport inhibition by iodide excess.

Thyroid hormones deficiencies during gestation increase the susceptibility to develop EAE in the offspring